We need the energy to live, grow, exercise, and survive. When we eat, in the digestive process, sugar is produced. Sugar finds its way from the digestive system into the blood, where insulin released from the pancreas helps break it down to release energy. This energy then becomes available to the body for its survival and other functions.
Excess sugar not required by us is converted into fats and stored in the liver. This stored fat is broken down into energy and made available when demand exceeds supply.
Insulin plays a blood sugar regulatory role. It prevents blood sugar levels from either rising too high. When blood sugar levels get too high, such a condition is called hyperglycemia. Hypoglycemia, on the other hand, is a low blood sugar condition.
Diabetes experts have indicated normal blood sugar levels for healthy people. According to the American Diabetes Association, the standard blood sugar level in a fasting state in adults should stay less than 100 mg/dl. A reading that is between 100 mg/dl and 126 mg/dl indicates that the individual is pre-diabetic. These are warning levels. They tell you that you on the verge of turning diabetic. The peak blood sugar levels for non-diabetics measured two hours after eating should not exceed 180 mg/dl.
Blood sugar levels, when these stay high, cause harm. But why do these levels rise at all? The inability of available insulin to fully break available blood sugar into energy leads to the rise in blood sugar levels.
Another reason is that the conversion process itself has become retarded. The cells of the body have become resistant to the available insulin. As a consequence, blood sugar levels rise. The medical name for such a condition is insulin resistance.
Type 2 diabetes is a lifestyle disease. Low levels of activity, excess consumption of carbohydrate and fat-rich foods, alcohol are some food that leads to a buildup of fats in the vascular system. This is one of the factors triggering type2 diabetes.
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